Publication date: 21 April 2020Source: Cell Reports, Volume 31, Issue 3Author(s): Emanuele Pignatti, Sining Leng, Yixing Yuchi, Kleiton S. Borges, Nick A. Guagliardo, Manasvi S. Shah, Gerard Ruiz-Babot, Dulanjalee Kariyawasam, Makoto Mark Taketo, Ji Miao, Paula Q. Barrett, Diana L. Carlone, David T. Breault
Publication date: 21 April 2020Source: Cell Reports, Volume 31, Issue 3Author(s): Adam Giess, Yamila N. Torres Cleuren, Håkon Tjeldnes, Maximilian Krause, Teshome Tilahun Bizuayehu, Senna Hiensch, Aniekan Okon, Carston R. Wagner, Eivind Valen
Publication date: 21 April 2020Source: Cell Reports, Volume 31, Issue 3Author(s): Wenxian Wu, Xiaojing Wang, Niklas Berleth, Jana Deitersen, Nora Wallot-Hieke, Philip Böhler, David Schlütermann, Fabian Stuhldreier, Jan Cox, Katharina Schmitz, Sabine Seggewiß, Christoph Peter, Gary Kasof, Anja Stefanski, Kai Stühler, Astrid Tschapek, Axel Gödecke, Björn Stork
Publication date: 21 April 2020Source: Cell Reports, Volume 31, Issue 3Author(s): Tito Borner, Hallie S. Wald, Misgana Y. Ghidewon, Bei Zhang, Zhidan Wu, Bart C. De Jonghe, Danna Breen, Harvey J. Grill
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Background/Aims: Hashimoto thyroiditis, characterized by positive thyroid peroxidase antibodies (TPOAbs), is caused by the interaction of genetic and environment factors. The aim of this study was to clarify the interaction of gene polymorphisms and iodine intake in the incidence of TPOAb positivity. Methods: 1,733 subjects were included in this study. Genomic DNA was extracted from peripheral blood white cells. Four SNPs (rs11675434 [TPO], rs3094228 [HCP5], rs9277555 [HLA-DPB1], and rs301799 [RERE])...
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